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. 2022 Apr 6;5:326. doi: 10.1038/s42003-022-03251-w

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a–c PCYT1A expression level was down-regulated by GSTM2 KD and up-regulated by GSTM2 OE; *p < 0.05, **p < 0.01. d PCYT1A KD induced LD expansion. e, f GSTM2 affected recruitment of GPAT4 and DGAT2 on LD surface (in the presence of oleic acid medium), bar, 10 μm, 1 μm (inlay). g Expression level of GPAT4 and DGAT2 was regulated by treatment of palmitic acid (ASK1 activator) and GS-4997 (ASK1 activity inhibitor). h Recruitment of GPAT4 on LD surface was regulated by palmitic acid or GS-4997 treatment, bar, 10 μm, 1 μm (inlay). i, j qPCR detection of PCYTA1 expression with PA or GS-4997 treatment respectively.