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. 2022 Mar;10(5):262. doi: 10.21037/atm-22-407

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2022 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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Molecular characterization of CAFs in PDAC. (A) Heatmap showing the top differently expressed genes in each CAF subtype. (B-D) KEGG pathway annotation of gene signatures of myofibroblastic CAF (B), inflammatory CAF (C) and antigen-presenting CAF (D). (E) Heatmap plotting the Pearson correlation between CAF subtype signature and hallmark signatures in a TCGA-PDAC cohort. R value >0.7 or <−0.3 are marked with asterisk (*). CAFs, cancer-associated fibroblasts; PDAC, pancreatic ductal adenocarcinoma.