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. 2022 Feb 9;477(5):1329–1338. doi: 10.1007/s11010-022-04369-5

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — Treatment of IL-1R antagonist inhibits TGF-β-mediated TGF-β accumulation in LX-2 cells. LX-2 cells were untreated (―) or pre-treated with 1 μg/ml of IL-1R antagonist for 1 h and then treated with 10 ng/ml of TGF-β1 for 24 h in the presence of IL-1R antagonist. a Cell lysate was prepared, and TGF-β expression was analyzed by western blotting. β-Actin was used as an internal control. b The amount of TGF-β released into the supernatant was quantified using an ELISA assay kit and normalized to the amount of total protein. *p < 0.01. (n = 3/group)