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. 2022 Apr 8;12:5944. doi: 10.1038/s41598-022-09963-7

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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A schematic diagram of NFκB (RelA)-hnRNPD axis in oral cancer. Transcription factor NFκB (Rel A) binds to its binding motifs in hnRNPD promoter region and mediates its transcriptional upregulation in oral cancer cells. Inhibition of NFκB by PDTC leads to decrease in hnRNPD protein and transcript levels by inhibition of NFκB (RelA)-hnRNPD axis in oral cancer (image created with Biorender.com).