. Author manuscript; available in PMC: 2023 May 1.

Published in final edited form as: Brain Behav Immun. 2022 Feb 22;102:224–236. doi: 10.1016/j.bbi.2022.02.024

Table 3.

Results of Models Relating Maternal Metabolic State and Offspring Microglial Counts in Amygdala Subregions

	Total			Lateral			Basolateral-dorsal			Basolateral-intermediate
Direct Effects	β(SE)	P	95 % CI of β	β(SE)	P	95 % CI of β	β(SE)	P	95 % CI of β	β(SE)	P	95 % CI of β
Maternal WSD → Amygdala region	−.443(.292)	.129	−.925, .038	−.554(.207)	.007	−.336, .196	−.483(.226)	.033	−.855, −.111	−.368(.252)	.144	−.782, .046
Pre-Pregnancy Adiposity → Amygdala region	.433(.269)	.100	.000, .886	.628(.166)	.001	.354, .901	.471(.215)	.029	.117, .824	.521(.206)	.011	.182, .860
Gestation Length → Amygdala region	.114(.092)	.215	−.037, .264	.164(.077)	.034	.036, .291	.226(.080)	.005	.095, .358	.138(.096)	.153	−.021, .297
Indirect Effect Paths
Maternal WSD → Pre-Pregnancy Adiposity → Amygdala region	.155(.107)	.145	−.020, .331	.219(.094)	.016	.069, .369	.166(.091)	.068	.016, .315	.183(.094)	.050	.029, .336

Note: WSD = Western Style Diet; Bold numbers indicate significance at p<0.05.