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. 2022 Mar 26;11(7):1835. doi: 10.3390/jcm11071835

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© 2022 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Study design. A total of 99 patients with CLD were included in this study. None of the included patients had any history of coagulopathy, sepsis, or platelet disorders. We investigated the relationship(s) among ADAMTS13:AC, VWF:Ag, and parameters related to Et in patients with CLD to determine the cause of the imbalance between ADAMTS13:AC and VWF:Ag in these patients. CLD, chronic liver diseases; ADAMTS13, a disintegrin-like and metalloproteinase with thrombospondin type 1 motifs 13; ADAMTS13:AC, ADAMTS13 activity; VWF, von Willebrand factor; VWF:Ag, VWF antigen; Et, endotoxins.