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. 2022 Mar 28;13:856713. doi: 10.3389/fimmu.2022.856713

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Copyright © 2022 Schrottmaier, Schmuckenschlager, Pirabe and Assinger

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Direct platelet-virus interactions. Platelets express a plethora of surface receptors to directly bind various virus families. Subsequent virus internalisation may occur via the cell surface or the open canalicular system. ACE2, Angiotensin-converting enzyme 2; CAR, Coxsackie and Adenovirus receptor; CCR, C-C chemokine receptor; CLEC-2, C-type lectin receptor 2; CR2, Complement receptor 2; CXCR, C-X-C chemokine receptor; DC-SIGN, Dendritic cell-specific intercellular adhesion molecule-3-grabbing non-integrin; GP, Glycoprotein; HSP, Heparan sulfate proteoglycan; HTNV, Hantaan virus; IAV, Influenza A virus; EMMPRIN, extracellular matrix metalloproteinase inducer; EV, Extracellular vesicle; NS1, Non-structural protein 1; PUUV, Puumala virus; RAV, Rotavirus A; TAT, Trans-activator of transcription; TLR, Toll-like receptor; TMPRSS2, Transmembrane protease serine subtype 2.