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. 2022 Apr 12;8:34. doi: 10.1038/s41421-022-00377-3

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a The G2/M-phase microglia of naive uninjured Cx3cr1-creER-yfp/+; Tnfaip3 fl/fl A20 cKO animals exhibited different gene expression profile from the G2/M phase microglia of control Cx3cr1-creER-yfp/+; +/+ animals 2 days after nerve injury. b The G2/M-phase microglia of naive uninjured Cx3cr1-creER-yfp/+; Tnfaip3 fl/flA20 cKO animals shared similar gene expression profile with the G2/M-phase microglia of control Cx3cr1-creER-yfp/+; +/+ animals 3 days after nerve injury. c, e The gene expression profile of the G2/M-phase microglia from Cx3cr1-creER-yfp/+; Tnfaip3 fl/fl A20 cKO animals remained largely unchanged 1 day (c), 2 days (d), or 3 days (e) after nerve injury.