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. 2022 Apr 12;13:1968. doi: 10.1038/s41467-022-29591-z

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a Association of Rtt106 with the promoter of the histone gene pair HTA1-HTB1, validating ChIP-seq analysis of Rtt106-6HA performed in this study. RTT106-6HA cells in log phase were collected for ChIP-seq analysis. Input, sequence reads of the sonicated genomic DNA before ChIP. Rtt106 ChIP, sequence reads of ChIPed DNA with Rtt106-6HA. b ChIP-seq analysis of Rtt106 showing association of Rtt106 with the promoter of the PDR5 gene. Positions of PDRE indicated by magenta lines. PDRE PDR responsive element. c The top 10 peaks of Rtt106 binding at promoter regions. Enrichment of Rtt106 at promoters was calculated as ChIP sequence reads normalised by Input sequence reads in a region spanning 500 bp upstream of the transcription start site of each gene.