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Depletion of STAT3 reverses KAT6B-regulated viability and apoptosis of glioma cells. (a)–(e) The U251 and LN229 cells were treated with ferroptosis activator erastin (5 μM) for 24 hours or treated with erastin (5 μM) for 24 hours followed by the treatment of KAT6B overexpressing plasmid and STAT3 shRNA. Cell viability (a) and colony formation number (b) and apoptosis (c)–(e) were analyzed by CCK-8, colony formation assay, and flow cytometry. ∗∗p < 0.01.
