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. 2022 Mar;10(6):344. doi: 10.21037/atm-22-982

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2022 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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Overexpression of miR-146a dramatically suppressed inflammation in TGF-β-induced thymocytes. TGF-β induced thymocytes were transfected with miR-146a NC, miR-146a inhibitor, or miR-146a mimics, respectively. (A) Changes in miR-146a and FOXP3 expressions in thymocytes were detected by RT-qPCR assay in each group. (B-E) ELISA assay displayed the changes of TNF-α, IL-10, IL-4, TGF-β levels in the transfected thymocytes. *P<0.05; **P<0.01. NC, negative control; TNF-α, tumor necrosis factor-α; IL-10, interleukin 10; IL-4, interleukin-4; TGF-β, transforming growth factor-β.