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. 2022 Jan 7;25(3):515–526. doi: 10.1007/s10120-021-01275-5

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — The effect of Tranilast on tumor cell apoptosis in mouse peritoneal and subcutaneous tumors. Representative image of immunohistochemical analysis of apoptosis using TUNEL and active caspase-3 immunofluorescence (original magnification × 100). The number of apoptotic cells in peritoneal tumors (a) and subcutaneous tumors (b) was measured and are shown as the average count of five non-overlapping tumor areas. Results are expressed as the mean ± SEM (n = 6 in each group for peritoneal tumors and n = 6 in each group for subcutaneous tumors, *p < 0.05, **p < 0.01)