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. 2023 Jan 4;102(8):814–825. doi: 10.1038/s41374-022-00783-x

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© 2022 United States & Canadian Academy of Pathology.

Since January 2020 Elsevier has created a COVID-19 resource centre with free information in English and Mandarin on the novel coronavirus COVID-19. The COVID-19 resource centre is hosted on Elsevier Connect, the company's public news and information website. Elsevier hereby grants permission to make all its COVID-19-related research that is available on the COVID-19 resource centre - including this research content - immediately available in PubMed Central and other publicly funded repositories, such as the WHO COVID database with rights for unrestricted research re-use and analyses in any form or by any means with acknowledgement of the original source. These permissions are granted for free by Elsevier for as long as the COVID-19 resource centre remains active.

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Fig. 3 — Characteristic histopathologic features of COVID-19-associated cardiac injury in critically ill patients.

Hematoxylin and eosin staining of representative cardiac injury among critically ill COVID-19 patients. COVID-19 patients exhibited pathological features of direct and indirect viral injury of the heart. Upper panels (A–D) are characteristic arteriole (A, B) and venous (C, D) thrombus. Regions of myocarditis from a COVID-19-positive decedent are shown in E, characteristic fibrin-rich thrombus and cardiomyocyte injury in COVID-19 patient (COVID-19) with history of myocardial infarction (F), mononuclear predominant inflammatory infiltrate within the visceral epicardium, and surrounding adipocytes (G) and a zone of ischemia (yellow arrows) in a patient with COVID-19-associated acute coronary syndrome demonstrating late healing with pigmented macrophages (H). Images A, C, F, and G are shown at ×10 magnification, B and E are shown at ×20 magnification, D is shown at ×40 magnification, and G is shown at ×4 magnification.