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. 2022 Apr 23;22:155. doi: 10.1186/s12890-022-01890-7

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Haplotype blocks for the asthma-associated locus. This figure shows the Manhattan plot a for the meta-analysis including the Saguenay–Lac-Saint-Jean asthma familial cohort (SLSJ cohort) and the Canadian Cohort Obstructive Lung Disease (CanCOLD) for the 2p24.3 COPD locus, and b haplotype blocks formed by the 4 SNPs associated with asthma in the 2p24.3 COPD locus using genotypes from the SLSJ cohort (done with Haploview software). The color of squares illustrates the strength of pairwise linkage disequilibrium (LD) values on a red and white scale where red indicates perfect LD (r² = 1.00) and white perfect equilibrium (r² = 0)