Skip to main content
. 2022 Mar 25;9(2):94–99. doi: 10.3390/dermatopathology9020012

Table 1.

EAH and the clinical differential diagnoses of EAH, including the features of demography and histopathological findings.

Disease Number of Cases
Reported in English
Histopathological Finding Reference
EAH <100 the lesion affects mainly mid and deep dermis and is composed of lobules of proliferating capillaries intricately admixed with sweat glands and ducts, fat and myxoid tissue. [12]
Eccrine nevus 20 It is characterized by groupings of normal to enlarged eccrine structures. (The absence of angiomatous hyperplasia) [13]
Tufted angioma 200 multiple, scattered lobules of small capillary type vessels with small oval to spindle shaped cells throughout the dermis and subcutaneous tissue imparting a “cannonball” or glomerular appearance. [14]
Smooth muscle hamartoma <20 (Only acquired type) It shows disseminated proliferation of mature smooth muscle cells of a central cigar-shaped nucleus and fibrillary and eosinophilic cytoplasm. [15,16]
Blue rubber bleb nevus syndrome 200 cutaneous lesions are non-specific and have features of venous malformations. Large, tortuous, dilated vessels with a single endothelial lining are noted, and smooth muscle may be present in the vessel walls. [17]