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. 2022 Apr 8;8:723332. doi: 10.3389/fcvm.2021.723332

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Copyright © 2022 Xu, Gu, Bian, Xu, Xia, Liu, Jia, Gu and Zhang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Remote conditioning by rhythmic compression decreases infarct size and ameliorates ventricular dysfunction in a rat model of AMI. (A) Schematic showing the overall animal study design used to test the cardioprotection of remote conditioning in a rat model of AMI. (B) Presentation of remote conditioning on forelimbs of rats. (C,D) TTC staining representing infarct area on multiples slices of an infarct rat heart and quantitative analyses of infarct size after 3 days of remote conditioning, N = 6. (E,F) Serum concentration of CK and cTnI of each group were evaluated after 3 days of remote conditioning, N = 3. (G–K) Representative M-mode echocardiographic images showing LV wall motion of the hearts and LVEF, LVFS measured by echocardiography at 6 h (baseline) and 3 days (endpoint) after MI, N = 6. *p < 0.05, ***p < 0.001, ****p < 0.0001 vs. CON.