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. 2022 Apr 29;12:7065. doi: 10.1038/s41598-022-11045-7

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Processes and pathways related to alveolar-capillary barrier function in atelectasis. Functional analysis of proteomics data showed the negative enrichment for processes or pathways related to alveolar-capillary barrier function in LPS(−) and LPS(+) conditions. Without LPS, atelectasis was associated with negatively enriched epithelial and endothelial processes, as well as Hippo signaling pathway. With LPS, the negatively enriched processes in atelectasis were involved in cell adhesion and junction. ECM organization and TGF-β signaling were negatively enriched in LPS-exposed and unexposed atelectasis. LPS lipopolysaccharide, ECM extracellular matrix, TGF transforming growth factor.