Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2022 Feb 24;20(5):812–832. doi: 10.1111/pbi.13784

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© 2022 The Authors. Plant Biotechnology Journal published by Society for Experimental Biology and The Association of Applied Biologists and John Wiley & Sons Ltd.

This is an open access article under the terms of the http://creativecommons.org/licenses/by-nc-nd/4.0/ License, which permits use and distribution in any medium, provided the original work is properly cited, the use is non‐commercial and no modifications or adaptations are made.

PMC Copyright notice

Model of the cellular mechanisms involved in gametophytic self‐incompatibility (GSI) in poppy (Papaver rhoeas). In an incompatible reaction, the pistil S1 protein binds to the pollen S1 receptor triggering an intracellular change in calcium concentration. This causes the rapid modification of two targets: Pr‐p26 shows an increase in phosphorylation leading to inhibition of its sPPase activity, and the actin cytoskeleton is reorganised and depolymerized. Both are predicted to cause rapid arrest of tip growth. Following this growth arrest, p56‐MAPK is activated and emits a signal to the PCD. PCD is linked to cytochrome C caspase activity and DNA fragmentation. This ensure that incompatible pollen do no start growing again. Adapted from McClure and Franklin‐Tong (2006).