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. Author manuscript; available in PMC: 2022 May 3.
Published in final edited form as: Clin Cancer Res. 2021 Jan 7;27(6):1695–1705. doi: 10.1158/1078-0432.CCR-20-4073

Table 1.

RTK fusions: Fusion partners and additional details for each of the 12 RTK fusion cases identified in our cohort

Fusion oncogene Fusion partner Known fusion partner Validation method Validation result Mismatch Repair Status Method of detection MSS/MSI status MLH1 promoter hypermethylation (Y/N) Site of primary tumor Stage at diagnosis Histology
RET NCOA4 Yes - - Deficient IHC and PCR Yes Ascending I Signet-ring-cell
RET NCOA4 Yes - - Deficient IHC and PCR Yes Descending II NA
NTRK1 TPR Yes IHC and Archer Dx Confirm Deficient IHC Yes Ascending IV Mucinous features
NTRK1 LMNA Yes IHC Confirm Deficient IHC Yes Ascending II Mucinous
NTRK1 LMNA Yes IHC and Archer Dx Confirm Deficient IHC Yes Ascending II Medullary
NTRK1 LMNA Yes IHC Confirm Deficient IHC and PCR Yes Cecum III Medullary features
ALK CAD Yes FISH Confirm Proficient IHC and PCR NA Cecum IV Poorly differentiated
ALK EML4 Yes - - Deficient IHC Yes Ascending IV Medullary and signet-ring-cell features
BRAF ARMC10 Yes - - Deficient IHC Yes Ascending III Poorly differentiated
FGFR2 TACC2 Yes Archer Dx Confirm Proficient IHC and PCR NA Rectosigmoid IV NA
ROS1 GOPC Yes - - Proficient IHC and PCR NA Rectosigmoid IV Mucinous features
NRG1 KIF13B Yes Archer Dx Confirm Proficient IHC NA Unknown IV NA

FISH: Fluorescence in situ hybridization. IHC: Immunohistochemistry. - : Tissue not available for further testing.