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Schematic illustration of the mechanisms by which edaravone dexborneol afforded neuroprotection against SAH-induced brain injury. SAH could cause neuronal apoptosis by inducing oxidative stress injury. Edaravone dexborneol treatment attenuates neuronal apoptosis by affording powerful antioxidant capacity after SAH, accompanied by the inhibition of lipid peroxidation products such as 4-HNE and MDA.
