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. 2022 May 6;12:190. doi: 10.1038/s41398-022-01959-1

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 2 — Venn Diagrams of gene overlap of A DE genes at FDR < 0.25 in caudate nucleus (CN), putamen (PUT), and ventral striatum (VS), B genes forming WGCNA expression-modules showing the strongest association with AUD status for CN, PUT, and VS, C genes forming WGCNA expression-module “e-CN-magenta” and those forming the methylation-modules “m-CN-red” and “m-CN-midnightblue”. Network plots depicting the WGCNA gene expression module hub genes showing the strongest association with AUD: D module magenta from caudate nucleus, E module black from putamen, and F module pink from ventral striatum.