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. 2022 May 9;29:29. doi: 10.1186/s12929-022-00812-3

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Myricetin exhibits cell cytotoxicity against androgen-dependent and -independent PCa cells. A Cells (normal immortalized RWPE-1, androgen-dependent LNCaP, and androgen-independent C4-2B, CWR22Rv1) were treated with different concentrations of myricetin over 3 days as indicated, followed by MTT assay. B H3K9me3 and H3K36me3 levels in myricetin-treated C4-2B cells for 2 days. The H3K9me3, H3K36me3, and H3 signals were detected in cell lysates by western blot analysis. M myricetin