Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2022 Apr 26;10:824075. doi: 10.3389/fcell.2022.824075

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

Copyright © 2022 Abudula, Fan, Zhang, Li, Zhou and Chen.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

PMC Copyright notice

ESCs-derived exosomes induced migration, angiogenesis, and upregulated inflammatory. (A) Wound-healing assay to verify the effects of exosomes on normal endometrial stromal cells (NSCs) migration. (B) Transwell assay to evaluate the migration ability, along with the quantification of migrated cells and migration rate. (C) Western blotting showed ectopic exosomes overexpressed MMP9, but there were no changes in the expression level of EMT- related proteins. (D) A tube formation assay along with quantification of the number of loops formed and loop length/per loop. (E) Western blotting analysis assessed the expression of VEGFR2, p-VEGFR2, PDGFR and p-PDGFR. (F) RT-qPCR tested the expression of inflammatory cytokines, data were expressed as mean ± SEM, NS, no significance, ****p < 0.0001.