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. 2022 May 10;41:171. doi: 10.1186/s13046-022-02374-6

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 8 — Tumor xenograft studies and mechanism diagram. A Left panel, the nude mice carrying subcutaneous xenografts in different groups and the dissected tumor samples from respective groups. Right panel, subcutaneous tumor growth curves. Data are presented as the mean ± SD (n = 5, each group). ^***P < 0.001 vs. OV-NC group; ^#P < 0.05, ^##P < 0.01 vs. OV-ZCRB1+OV-circHEATR5B+OV-HEATR5B-881aa group by two-way ANOVA. B Survival curves of nude mice with orthotopic xenografts (n = 5, each group). C The schematic of the mechanisms of the ZCRB1/circHEATR5B/HEATR5B-881aa/JMJD5/PKM2 pathway in GBM cells