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Schematic diagram of the molecular mechanism by which curcumin promotes antioxidant activity of intracranial microglia after intracerebral hemorrhage. By activating the Nrf2/ARE pathway, curcumin promoted the expression of many downstream antioxidant-related genes (including HO-1, NQO1, and Gpx4). Therefore, curcumin inhibited the cerebral oxidative injury after ICH.
