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BBR induced NSCLC cell apoptosis via the activation of the ASK1/JNK pathway in vivo. (A) IHC analysis of the expression of phosphorylation-ASK1, phosphorylation-JNK, CL-caspase-3, and Bax in the tumor tissues treated with BBR (500 mg/kg). (B) The protein levels from (A) were quantified. ***P<0.001. BBR, berberine; ASK1, apoptosis signal-regulating kinase 1; JNK, c-jun-NH2-kinase; NSCLC, non-small cell lung cancer; IHC, immunohistochemical.
