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. 2021 Dec 9;220(2):iyab212. doi: 10.1093/genetics/iyab212

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© The Author(s) 2021. Published by Oxford University Press on behalf of Genetics Society of America.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (https://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited.

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Components of environmental condition-responsive signaling pathways control DNA site-dependent recombination hotspots in cis and in trans. The effects of each indicated protein on the activation of each class hotspots was measured; proteins that bind directly to each hotspot DNA site are highlighted (bold). Relative rates of recombination in null mutant vs wild-type are grouped by class of hotspot: (A) M26 DNA site; (B) CCAAT DNA site; (C) Oligo-C DNA site. Data are mean ± SD from at least three independent biological replicates; differences with P ≤ 0.05 (*) are indicated for mutant vs wild-type.