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. 2022 May 12;13(5):452. doi: 10.1038/s41419-022-04903-9

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 8 — Autophagy signaling pathway molecule ATG7 was analyzed using KLF2-ChIP and quantitative PCR methods after the addition of ferutinin to the DPSCs for 24 h. Active marks of the ATG7 gene for both H3K4me3 and H3K27Ac molecules were evaluated in the promoter site using two different primers such as A ATG7-1 and B ATG7-2, and shown graphically. Vehicle-treated DPSCs were considered as a control. Star (*) indicates a statistical significance (p < 0.05) when compared ferutinin-treated cells to the controls (n = 3).