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. 2022 Apr 29;12:873532. doi: 10.3389/fonc.2022.873532

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Copyright © 2022 Khadse, Haakensen, Silwal-Pandit, Hamfjord, Micke, Botling, Brustugun, Lingjærde, Helland and Kure

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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(A) Oncoplot showing mutation status in EGFR and KRAS (red: mutated, green: wild-type, gray: missing), loss of heterozygosity (LOH) at the KRAS locus (red: LOH, green: no LOH), and genomic instability index (GII) in the Oslo cohort (red: high, blue: low). (B) KRAS expression by KRAS LOH status indicating higher expression in samples with no LOH in KRAS. (C) KRAS expression by KRAS mutation status indicating high expression in samples with KRAS mutation (wt: wild-type; mut: mutation). (D) KRAS expression by KRAS LOH status grouped by KRAS mutation status in lung adenocarcinomas with respect to KRAS LOH and mutation status [0: wild-type (blue); 1: mutation (yellow)]. NA, Not available.