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. 2022 May 13;23:177. doi: 10.1186/s12859-022-04726-7

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 7 — Comparison of immune status between the high-risk and low-risk groups. A, B Comparison of the stromal scores, immune scores, ESTIMATE scores, and tumor purity. (A TCGA cohort; B ICGC cohort). C The differences of the immune cell subtypes between the two risk groups in the TCGA cohort. D The differences of the immune-related pathways between the two risk groups in the TCGA cohort. E The differences of the immune cells subtypes between the two risk groups in the ICGC cohort. F The differences of the immune-related pathways between the two risk groups in the ICGC cohort. G, H Comparison of mRNAsi and mDNAsi of the high-risk and the low-risk groups in the TCGA cohort. (G mRNAsi; H mDNAsi). I, J The relationship of mRNAsi and m DNAsi with the risk score in the TCGA cohort. (I mRNAsi; J mDNAsi) (*P < 0.05, **P < 0.01, *** P < 0.001, ns P > 0.05)