Figure 2.

Immune responses and overall intracellular events triggered in the face of CoV-2. In normal conditions, immune cells enter the alveoli of the lungs through the blood. When CoV arrives in the alveolar compartment, these immune cells oppose them and eventually produce cytokines. Positively regulating, cytokines trigger more immune cells, eventually producing more cytokines. During these events, the fibrin of the alveoli increases, resulting in partial destruction and increased permeability of the alveoli. Consequently, fluid goes on the battlefield (alveoli) through the capillaries, causing destruction and edema of the infected lung. However, when a virus can compete with these challenges, it will be able to enter the host cell. In the host cell, the viral genome is generally released and the host equipment is used to replicate the viral particles. The following sections of the article provide more details in this regard.