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. 2022 May 13;22:218. doi: 10.1186/s12872-022-02648-z

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© The Author(s) 2022

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Example images of MV, GLS and myocardial perfusion in STEMI patients with normal (A) or impaired (B) MVP. The IRA of both patients is left anterior descending artery (LAD). Patient A had normal MVP but patient B had impaired MVP in the apex (arrows). Compared with patient A, patient B had significantly reduced GWI (415 mmHg% vs. 1120 mmHg%), GCW (636 mmHg% vs. 1309 mmHg%), GWE (55% vs. 93%) and GLS (absolute value 6% vs. 15%), but larger GWW (384 mmHg% vs. 80 mmHg%)