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. 2022 May 3;12:894836. doi: 10.3389/fcimb.2022.894836

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Copyright © 2022 Li, Wu, Luo, Niu, Yan, Fang, Ke, Chen, Xu, Li, Hu, Cao, Chen, Tu and He

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Mutational signatures in HBV-positive and HBV-negative samples. (A) Genetic profiles of seven HBV-positive and nine HBV-negative gastric cancer (GC) samples. (B) Comparison of the most frequently mutated genes between the HBV-positive and HBV-negative GC samples. (C) Expression analysis of KMT2B, FGF12, and TUBB2B in 32 pairs of GC and non‐tumor tissues from The Cancer Genome Atlas (TCGA) database. (D) Overall survival based on the expression levels of KMT2B, FGF12, and TUBB2B in 346 patients with GC from TCGA database by Kaplan-Meier curves analysis; (E) Gene Ontology annotation analysis of frequently mutated genes in GC (mutation frequency ≥ 2, P < 0.05). HBV, hepatitis B virus; HBV-positive, positive for HBsAg or anti-HBs, anti-HBe, and anti-HBc; HBV-negative, negative for HBsAg, HBeAg, anti-HBe, anti-HBs, and anti-HBc. ***P < 0.001.