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. 2022 May 18;12:8319. doi: 10.1038/s41598-022-12406-y

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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(a)–(e) Photomicrograph of H&E-stained liver sections from the indicated groups: (a) control male mice, showing the central vein (cv) and obvious appearance of Kupffer cells (blue arrow); (b and c) AC group mice, revealing loss of hepatic architecture, hemorrhage (yellow arrow), inflammation (red arrow), fragmented nuclei (black arrow), multiple ballooned hepatocytes, and vacuolated cytoplasm; (d): AC + Cur group , showing hepatic tissues with more modest cell infiltration (red arrow); (e): AC + NCur group, demonstrating healthy architecture appearance similar to control (H&E Bar = 50 µm). (f) Liver histopathology scores of groups (mean ± SE) and values with different letters are significantly different (p < 0.001).