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. Author manuscript; available in PMC: 2023 Jul 1.
Published in final edited form as: J Comp Neurol. 2022 Feb 28;530(10):1729–1738. doi: 10.1002/cne.25310

Table 4.

ROI-based correlations of FA and depressive symptoms, controlling for age and sex

BDI-II Factor ROI Multiple Regression
Effect Estimate (se) p- value
Dysphoric mood Fornix (cres) / Stria terminalis R 0.0070 (0.0032) 0.037
Loss of interest/pleasure Splenium of corpus callosum 0.0262 (0.0126) 0.048
Somatic Corticospinal tract L 0.0121 (0.0056) 0.041
Superior cerebellar peduncle R 0.0122 (0.0051) 0.024
Cerebral peduncle R 0.0140 (0.0063) 0.034
Anterior corona radiata R 0.0167 (0.0068) 0.021
External capsule R 0.0137 (0.0052) 0.014
External capsule L 0.0139 (0.0054) 0.015
Cingulum (cingulate gyrus) R 0.0150 (0.0052) 0.008
Cingulum (cingulate gyrus) L 0.0133 (0.0055) 0.024
Cingulum (hippocampus) L 0.0193 (0.0082) 0.026
Fornix (cres) / Stria terminalis R 0.0124 (0.0052) 0.024
Superior fronto-occipital fasciculus R 0.0140 (0.0059) 0.026
Superior fronto-occipital fasciculus L 0.0158 (0.0056) 0.009
Tapetum R 0.0174 (0.0059) 0.006
Tapetum L 0.0150 (0.0046) 0.003

The subcallosal cingulate cortex (SCC) and its connections have been implicated in the pathophysiology of major depressive disorder (MDD). We used diffusion tensor imaging to examine the MDD-related structural abnormalities underlying depressive symptoms in PD (n=31). SCC (green box) and medial prefrontal cortex (yellow box) connectivity (group averaged red tract) in the left hemisphere was positively correlated with severity of dysphoric mood symptoms, suggesting abnormal SCC connectivity underlies depressive symptoms in both PD and MDD, particularly when depression in PD is dominated by dysphoric mood.