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. 2022 Apr 1;53(6):2006–2015. doi: 10.1161/STROKEAHA.121.035826

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© 2022 The Authors.

Stroke is published on behalf of the American Heart Association, Inc., by Wolters Kluwer Health, Inc. This is an open access article under the terms of the Creative Commons Attribution Non-Commercial-NoDerivs License, which permits use, distribution, and reproduction in any medium, provided that the original work is properly cited, the use is noncommercial, and no modifications or adaptations are made.

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Figure 2. — Shows the individual data points for vasomotor reactivity parameters at baseline and follow-up. Blood-oxygen-level-dependent (BOLD) amplitude (A), time to peak (B) and time to baseline (C) are shown. Presymptomatic Dutch-type hereditary cerebral amyloid angiopathy (D-CAA) mutation carriers are represented in blue, symptomatic mutation carriers in red, and controls in green. Significant longitudinal differences are shown with an asterisk.