Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2022 May 10;12:880636. doi: 10.3389/fcimb.2022.880636

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

Copyright © 2022 Wu, Jian, Tang, Liu, Liu, Liu and Wu

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

PMC Copyright notice

Genetic and transcriptional alterations of HP-related genes in stomach adenocarcinoma (STAD). (A) Mutation frequencies of 151 HP-related genes in 433 patients with STAD from the TCGA cohort. (B) Locations of CNV alterations in HP-related genes on 23 chromosomes. (C) The interaction between 73 HP-related genes in STAD. Red semicircles represent the gene is HP infection-associated. Purple and green semicircles represent risk factors and favorable factors, respectively. The circle size represented the effect of each regulator on the prognosis, and the range of values calculated by Log-rank test was p < 0.0001, p < 0.001, p < 0.01, p < 0.05 and P < 1, respectively. The line connecting the regulators represents their interaction, with the line thickness indicating the strength of the association between the regulators. Pink and blue stand for positive and negative correlation with statistical significance respectively. (D) Frequencies of CNV gain, loss, and non-CNV among HP-related genes.