Skip to main content
. Author manuscript; available in PMC: 2022 May 24.
Published in final edited form as: Nat Metab. 2020 Sep 14;2(9):849–857. doi: 10.1038/s42255-020-00277-4

Figure 1:

Figure 1:

Exercise-induced protection from tissue-specific perturbations in organs involved in cancer regulation or prone to malignancy. Blue boxes contain illustrative examples in the liver, colon and bone marrow. Grey boxes indicate examples of tissues where data to support exercise-induced regulation of tissue biology in the absence of frank malignancy is currently lacking. NAFLD: non-alcoholic fatty liver disease; AMPK: adenosine monophosphate kinase; NFκB: nuclear factor kappa-light-chain-enhancer of activated B cells; TNFα: tumour necrosis factor alpha; IL: interleukin: IL; KC: keratinocyte chemoattractant; MCP-1: monocyte chemoattract protein; COX2: cyclooxygenase-2; HSPC: hematopoietic stem and progenitor cell; Cxcl12: C-X-C motif chemokine 12.