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. 2022 May 11;9:854554. doi: 10.3389/fcvm.2022.854554

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Copyright © 2022 Gupta, Shrivastava, Vijayvergiya, Chhikara, Datta, Aziz, Singh Meena, Nath and Kumar.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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OCT in in-stent restenosis (ISR). (A) Homogenous neointimal hyperplasia (NIH) in bare metal stent (BMS) restenosis. (B) Heterogenous NIH in a drug-eluting stent (DES) ISR. (C,D) Showed neoatherosclerosis as a cause of ISR in a well-expanded stent with well-demarcated calcium and fibrotic ingrowth. (E–H) Coronary angiography showing proximal LAD bioresorbable vascular scaffolds (BVS) ISR after 2 years of implantation. (F) The OCT image shows a distal marker of BVS, (G) almost completely absorbed BVS struts in the middle and distal part of the stent, and (H) BVS ISR at the proximal segment with peri-strut low intensity areas.