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. 2022 May 24;11(1):2075204. doi: 10.1080/2162402X.2022.2075204

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© 2022 The Author(s). Published with license by Taylor & Francis Group, LLC.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Figure 3. — NK, NKT, and γδ T cell surface markers in the TH-MYCN^+/+ TME. Cell surface protein expression from TH-MYCN^+/+ neuroblastoma-derived natural killer (NK), invariant natural killer T (NKT), and gamma delta (γδ) T Cells from 14G2a midpoint (Mid), 14G2a terminal (Term), Isotype, and PBS groups. (a) NK Inhibitory receptor expression including transforming growth factor beta receptor-1 (TGFBR-1), interleukin-15 receptor-alpha (IL-15Rα), Ly49A, and NKG2A. (b) NK Activation receptor expression of Ly49H and NKG2D. (c-e) Expression of markers of cellular activation CD38 and CD107a are shown for NK, NKT, and γδ T cells. Two-tailed Student’s t-test performed to compare groups with significance set as p ≤ .05 (trend p ≤ .10).