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. 2022 May 25;13(5):495. doi: 10.1038/s41419-022-04944-0

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — A, B) C57B6/L mice were subjected to 2/3 PHx and sacrificed at indicated time points. Eotaxin expression levels were evaluated by qPCR and ELISA. N = 3–6 mice for each group. C, D C57B6/L mice were injected with APAP (300 mg/kg) and sacrificed at indicated time points. Eotaxin expression levels were evaluated by qPCR and ELISA. N = 3–6 mice for each group. E, F Primary murine hepatocytes were treated with HGF (20 ng/ml) and harvested at indicated time points. Eotaxin expression levels were evaluated by qPCR and ELISA. Error bars represent SD (*p < 0.05, one-way ANOVA). All experiments were repeated three times and one representative experiment is shown.