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. 2022 May 25;13(5):495. doi: 10.1038/s41419-022-04944-0

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — WT and Brg1^LKO mice were injected with adenovirus carrying eotaxin expression vector (Ad-Eotaxin) or control adenovirus (Ad-GFP) followed by 2/3 PHx. A Scheme of animal protocol. B, C Hepatic eotaxin expression was examined by qPCR and ELISA. D Liver weight versus body weight was calculated at 24 and 48 h post-surgery. E Expression levels of pro-proliferative genes in the liver samples collected at 48 h were examined by qPCR. F Paraffin embedded sections were stained with anti-Ki67. N = 5–8 mice for each group.