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. 2022 May 12;12:764621. doi: 10.3389/fonc.2022.764621

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Copyright © 2022 Song, Li, Chen, Zhang, Yang, Zhang, Song, Miao, Chang and Shi

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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The FAM222A-AS1 knockdown inhibits tumour growth in vivo, and overexpression of FAM222A-AS1 promote the tumour growth in mice. (A) HCT116 cells were stably transfected with the FAM222A-AS1-shRNA vector and injected subcutaneously into nude mice. Compared with the control group (-Dox), FAM222A-AS1 downregulation (+Dox) inhibited tumor growth. (B) SW620 cells were stably transfected with the FAM222A-AS1-shRNA vector and injected subcutaneously into nude mice. Compared with the control group (-Dox), FAM222A-AS1 downregulation (+Dox) inhibited tumor growth. (C) HT29 cells were stably transfected with the FAM222A-AS1-overexpression vector and injected subcutaneously into nude mice. Compared with the control group (-Dox), FAM222A-AS1 overexpression (+Dox) promote tumor growth. HT29-OE, HT29 stable cells transinfected with FAM222A-AS1 overexpression vectors. (*P < 0.05, **P < 0.01).