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editorial

. 2022 May 20;11(5):1012. doi: 10.3390/antiox11051012

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© 2022 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Hypothetical redox homeostasis and stress-response interaction scheme (adapted from [1,10,11,18,19]. AMPK, AMP-activated protein kinase; ATF6, activating transcription factor 6; ATM, ataxia-telangiectasia-mutated; ATR, ataxia-telangiectasia and Rad 3-related; FOXO, forkhead box protein; HIF, hypoxia inducible factor; HSF, heat shock factor; IRE1, inositol-requiring enzyme 1; NF-κβ, nuclear factor kappa-light-chain-enhancer of activated B cells; Nrf2, nuclear factor erythroid-2 related factor 2; SIRTs, sirtuins.