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. 2022 Apr 28;11(5):873. doi: 10.3390/antiox11050873

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© 2022 by the authors.

Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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Expression of inflammation-related genes (A) IL-1b, (B) IL-6, (C) TNF-a, (D) 5-LOX, (E) PSGT2, and oxidative-stress-related gene (F) GPX2, (G) NQO1, and (H) GCLC expression upon treatment with BBR-LCNs on 16HBE cells. Values are expressed as mean ± SEM (n = 4–6); * p < 0.05 ** p < 0.01, **** p < 0.0001 (control vs. 5%CSE) and ^## p < 0.01, ^### p < 0.001 ^#### p < 0.0001 (5% CSE vs. 5% CSE + BBR-monoolein). ns = not significant. Analysis was performed by a one-way ANOVA followed by Tukey multiple comparison test.

Expression of inflammation-related genes (A) IL-1b, (B) IL-6, (C) TNF-a, (D) 5-LOX, (E) PSGT2, and oxidative-stress-related gene (F) GPX2, (G) NQO1, and (H) GCLC expression upon treatment with BBR-LCNs on 16HBE cells. Values are expressed as mean ± SEM (n = 4–6); * p < 0.05 ** p < 0.01, **** p < 0.0001 (control vs. 5%CSE) and ^## p < 0.01, ^### p < 0.001 ^#### p < 0.0001 (5% CSE vs. 5% CSE + BBR-monoolein). ns = not significant. Analysis was performed by a one-way ANOVA followed by Tukey multiple comparison test.