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. Author manuscript; available in PMC: 2023 Jan 1.
Published in final edited form as: Semin Nephrol. 2022 Jan;42(1):29–43. doi: 10.1016/j.semnephrol.2022.01.005

Figure 3: Pathogenesis of recurrent primary FSGS.

Figure 3:

Left panel: Previous published reports have found that variables broadly related to recipients’ immune system status may predict recurrent FSGS. The association with inherited factors is even less defined at the moment. Right panel: Representative photomicrographs of recurrent primary FSGS (A) A normocellular glomerulus showing mild prominence of the podocytes on day 12 after transplantation in a patient presented with full nephrotic syndrome (periodic acid–Schiff, original magnification ×600) (B) This was associated with complete foot process effacement (electron microscopy, original magnification ×6,000) (C) Several segmental sclerotic lesion was noted in the follow-up biopsy, which was performed 3 months post-transplant (arrows, representative image, periodic acid–Schiff, original magnification ×600).