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. 2021 Feb 17;42(5):1557–1568. doi: 10.1007/s10571-021-01046-x

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Effects of EphA4 and ephrin-B ligands on cytokine secretion by damaged astrocytes. a ELISAs detected the expression of cytokines related to astrocyte function in the spinal cord of the epha4^−/− mice. b ELISAs examined the influence of EphA4 on the expression of cytokines related to astrocyte function in damaged astrocytes. c ELISAs examined the influence of EphA4 on cytokine secretion by damaged astrocytes with ephrin-B1 deletion. d ELISAs examined the influence of EphA4 on cytokine secretion by damaged astrocytes with ephrin-B2 deletion. e ELISAs examined the influence of EphA4 on cytokine secretion by damaged astrocytes with ephrin-B3 deletion. f Western blots confirmed the deletion of EphA4 and ephrin-B ligands in cultured astrocytes. N = 5; **P < 0.01