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. 2022 Jan 29;29(28):43114–43125. doi: 10.1007/s11356-021-18331-3

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — Effect of acetamiprid and azoxystrobin on testis tissue of rats. A Seminiferous tubules (ST) in the control group showed a normal appearance. A photomicrograph of seminiferous tubules (ST) of the treated group with acetamiprid. B Low dose demonstrated sloughing of germinal epithelium (S). C Medium dose demonstrated degenerated spermatogenic cells with vacuole formation (V). D High dose demonstrated degenerated spermatogenic cells with vacuole formation (V). Seminiferous tubules (ST) of the treated group with Azoxystrobin. E Low dose demonstrated degenerated spermatogenic cells with vacuole formation (V). F Medium dose demonstrated degenerated spermatogenic cells with vacuole formation (V). G High dose demonstrated degenerated spermatogenic cells with vacuole formation (V). (hematoxylin–eosin (H&E) stained, × 200)