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. 2022 May 28;13(5):506. doi: 10.1038/s41419-022-04908-4

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© The Author(s) 2022

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — A Putative complementary sites within miR-25-3p and circRNA BTG2 were predicted by bioinformatics analysis. B Dual-luciferase reporter assays demonstrate that miR-25-3p is a direct target of circRNA BTG2 in 293 T cells. C The Ago2 RIP showed that Ago2 significantly enriched circRNA BTG2 and miR-25-3p. D The expression level of circRNA BTG2 in 40 glioma tissues and matched para-carcinoma normal tissues was determined by qRT-PCR. E The expression level of miR-25-3p in the above tissues was determined by qRT-PCR. F The expression levels of miR-25-3p are negatively correlated with circRNA BTG2 in glioma tissues. **P < 0.01 and ***P < 0.001 for statistical differences.