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. 2022 May 30;12(5):84. doi: 10.1038/s41408-022-00677-7

Table 3.

Response outcomes in patients with R/R AML by prior TKI status according to FLT3 mutation type in the CHRYSALIS and ADMIRAL studies.

CHRYSALIS Trial: 120- or 200-mg Gilteritinib
Response parameter, n (%) Prior TKI No Prior TKI
FLT3-ITD (n = 29) FLT3-ITD and -TKD (n = 4) FLT3-ITD (n = 94) FLT3-TKD (n = 9) FLT3-ITD and -TKD (n = 7)
CR 1 (3) 0 16 (17) 0 0
CRi 8 (28) 2 (50) 20 (21) 1 (11) 4 (57)
CRp 3 (10) 0 7 (7) 0 0
PR 2 (7) 0 6 (6) 1 (11) 1 (14)
NR 13 (45) 2 (50) 34 (36) 7 (78) 1 (14)
NE 2 (7) 0 11 (12) 0 1 (14)
CRca 12 (41) 2 (50) 43 (46) 1 (11) 4 (57)
ADMIRAL Trial: 120-mg Gilteritinib
Response parameter, n (%) Prior TKI No Prior TKI
FLT3-ITD (n = 24) FLT3-TKD (n = 5) FLT3-ITD and -TKD (n = 4) FLT3-ITD (n = 191) FLT3-TKD (n = 16) FLT3-ITD and -TKD (n = 3)
CR 2 (8) 2 (40) 2 (50) 42 (22) 2 (13) 0
CRi 6 (25) 0 1 (25) 53 (28) 2 (13) 1 (33)
CRp 3 (13) 1 (20) 0 12 (6) 2 (13) 1 (33)
PR 5 (21) 0 0 23 (12) 5 (31) 0
NR 7 (29) 1 (20) 1 (25) 50 (26) 5 (31) 1 (33)
NE 1 (4) 1 (20) 0 11 (6) 0 0
CRca 11 (46) 3 (60) 3 (75) 107 (56) 6 (38) 2 (67)

Bold font indicates aggregate responses.

aDefined as the sum of patients who achieved CR, CRi, and CRc.

AML acute myeloid leukemia, CR complete remission, CRc composite complete remission, CRi complete remission with incomplete hematologic recovery, CRp complete remission with incomplete platelet recovery, ITD internal tandem duplication, NE not evaluable, NR no response, PR partial remission, R/R relapsed or refractory, TKD tyrosine kinase domain, TKI tyrosine kinase inhibitor.